Antispasmodic medication non sedating


10-Feb-2019 13:45

Spasmolytic agents generally work by either enhancing the level of inhibition, or reducing the level of excitation.

Inhibition is enhanced by mimicking or enhancing the actions of endogenous inhibitory substances, such as GABA.

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It may also reduce pain in patients by inhibiting the release of substance P in the spinal cord, as well.

Several of these agents also have abuse potential, and their prescription is strictly controlled.

receptors in the brain and spinal cord, resulting in hyperpolarization of neurons expressing this receptor, most likely due to increased potassium ion conductance.

Improving sleep is also beneficial for patients who have fibromyalgia.

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Because of the enhancement of inhibition in the CNS, most spasmolytic agents have the side effects of sedation, drowsiness and may cause dependence with long-term use.

The binding of two acetylcholine molecules results in a conformational change in the receptor that opens the sodium-potassium channel of the nicotinic receptor.